Hemp Hero Luke Scarmazzo

The USA Hemp Museum, Richard M. Davis, founder, curator, hempologist, is pleased to introduce you to a hemp hero, Luke Scarmazzo.

This courageous young hemp warrior has a few medical marijuana buyers clubs in California that provide safe medicine to people with doctor's notes in a state where the people already voted hemp legal.

The DEA reports about Luke's case:


"....Section 846-Conspiracy to Distribute Marijuana and Section 841(a)(1)-Distribution of Marijuana. The maximum statutory penalty for Conspiracy to Distribute Marijuana is a minimum mandatory term of imprisonment of ten years, and a maximum term of imprisonment of life; a $4 million dollar fine. The maximum statutory penalty for Distribution of Marijuana is a minimum mandatory term of imprisonment of ten years, and a maximum term of imprisonment of life; a $4 million dollar fine.

This case is the product of an extensive joint investigation conducted by the U.S. Drug Enforcement Administration, Modesto Police Department, Modesto Narcotics Enforcement Team (“MNET”), Stanislaus Drug Enforcement Agency (“SDEA”), and the Internal Revenue Service.

This case is being prosecuted by the United States Attorney’s Office, Eastern District of California, in Fresno. The charges are only allegations and the defendants are presumed innocent unless proven guilty beyond a reasonable doubt."


We are legislated to death. How does one prove themselves innocent when no real crime has been committed? How is it a crime to supply effective medicine to sick people for substantially less than prescription drugs?


If Luke owned or worked in a pharmacy, he could sell folks medicine that kills people. Instead, he chose to supply hemp medicine from which there is not one known case of death other than murder by government.


More on Luke in future posts, but by way of introduction, here are the webeos about Medical Marijuana on 60 Minutes that includes a discussion of his case and a webeo of his tune about being a hemp business man.


60 Minutes on Medical Marijuana Pt 1




60 Minutes On Medical Marijuana Pt 2




California Hemp Activist Music Video
Kraz-Business Man By Luke Scarmazzo






If you are in a position to support this hemp hero/warrior and other victims of government policy, please do so. They are on the front lines of this battle to save ourselves, and we hempsters must support our troops.


Hemp For Victory Now!

Physicians Desk Reference For Herbal Medicines - Hemp

From a typing of the section of the book Physicians Desk Reference For Herbal Medicines, the section on hemp's healing applications. Animal testing (not that there is any lack of patient and doctor testimony going back 5,000 plus years) revealed to the PDR what local healers have known all their lives. Hemp helps heal.

"Respiratory tract: The inhalation of Marihuana smoke caused bronchial dilation in healthy subjects. Methacholine-induced asthma attacks can be terminated by inhaling marihuana, in this case only psychomimetic cannaboids are active.

Eyes: The ability of cannabis products to reduce intra-ocular pressure was discovered accidentally during trials on the effect of inhaling high doses. During the tests, intra-occular pressure dropped by 45%. Eye drops applied locally had the same effect as standard medication but the effect lasted longer.

Immune system: In vitro and in animal testing, depending on the tissue, the immune system was significantly suppressed after cannaboid administration. Antimicrobial action: CBC CBDA, CBG and d-9-tetrahydrocannabinol displayed antibacterial effects. CBC and d-9-tetrahydrocannabinol are bacteriostatic and bactericidal against streptocci and staphylococci.

Tumor inhibiting effect: The in-vitro inhibiting effect of d-9-tetrahydrocannabinol, d-8-tetrahydrocannabinol, and CBN on the growth of transplanted lung tumors has been documented.

Heart, circulation: Cannaboids increase heart frequency, peripheral vasodilatation causes an increase in systolic blood pressure in the prone position and a decrease in the supine position.

Other effects: d-9-tetrahydrocannabinol is said to be an appetite stimulant. Long term usage leads to a clear increase in tolerance for most of the pharmacological effects."

Knowledge is power and with hemp, we can save ourselves. For more information, visit us at the USA Hemp Museum, a private museum with a virtual wing, Richard M. Davis founder, curator and author of HEMP FOR VICTORY: A GLOBAL WARMING SOLUTION.

Here's the entire piece with a great literature list at the end.

If able, please buy the book and share the information.

Physicians Desk Reference For Herbal Medicines
Pages 712-714

Cannabis Sativa—Marijuana

Description:

Flower and Fruit: Hemp is dioecious. The female flowers are reduced to the perigone with one bract. The complete inflorescences for a leafy, false spike. The male flowers form panicles rich in pollen. Pollination is by wind. The fruit is a gray-green, glossy achaene, 3.5 to 5 mm long and 2.5 to 4 mm wide. The seeds have little endosperm, are white, oily-fleshy and hooked.

Leaves, Stem and Root: The variety cannabis is an annual or biennial plant, which is usually branched and grows up to 5 m. The plant has erect, rough-haired and compressed bristles. The leaves are long-petioled and 3 to 7 pinnate. The leaflets are lanceolate and serrate.

Habitat: The plant probably originated in the Middle East. Today it is grown worldwide in temperate and tropical regions.

Production: Indian hemp is the dried flowering or fruiting branch tips of Cannabis sativa var. indica. Production depends on the origin. One method is by striping the leaves. Another method is stripping the resin exuded from the flowers and multiple fruit, which is shaped into balls or sheet forms. The final method involves cutting 5 cm to 10 cm long branch tips, which have just borne fruit, removing the leaves, pressing the shooting tips and gathering them into bundles.

Not To Be Confused With: Prior to being used as a narcotic, marijuana was often combined with Nicotiana tabacum, Lavandula officinalis, Nepeta catarina or Origanum vulgare. It is possible to confuse Marijuana with varieties of Urtica, Moraceae, Ulmaceae and Boraginaceae.

Other Names: Cannabis, Pot, Bhang, Grass, Indian Hemp, Weed, Ganja, Kif
ACTIONS AND PHARMACOLOGY—COMPOUNDS

Cannabinoids: chief active agent 9-tetrahydrocannabinol (9-THC 1-THC), in addition to 60 additional cannabinoids

Volatile oil: of a very complex composition, with, among other things beta-caryophyllenes, humules, caryophyllene oxide, alpha-pinenes, beta-pinenes, limonene, myrcene, beta-ocimene

Flavonoids

EFFECTS

Psychotropic action: In most subjects the effect is registered following an oral dose of 20 mg d-9-tetrahydrocannabinol or after inhaling a cigarette with 2% d-9-tetrahydrocannabinol. The symptoms are mood swings, reduction in drive, inability to think clearly, confusion, lack of concentration, impairment of short term memory and perception of time. Sensory impressions become heightened or experienced differently.

Complex tasks become more difficult, the capacity to understand or empathize is impaired. Negative reactions such as anxiety, panic and psychosis can occur.

It is only possible to describe this effect in animal tests, on the basis of free behavioral and controlled behavioral tests. A stimulating effect has also been observed with lower doses. Not all cannaboids cause the same effect. CBC, CBD and CBG have no psychomimetic effect. Various interactions occur in combination with d-9-tetrahydrocannabinol.

Antiemetic action: has been reported in clinical studies involving cancer patients receiving chemotherapy.

Anticonvulsive action: d-9-tetrahydrocannabinol reduces the clinical and electrographic convulsion intensity in cats.

Analgesic characteristics: d-9-tetrahydrocannabinol displays analgesic characteristics, while at the same time partially increasing sensitivity to pain.
Body temperature: In animal tests d-9-tetrahydrocannabinol and other cannaboids reduced body temperature. The maximum reduction was relatively small. A stronger hypothermic effect was observed in higher doses, which affected behavior.

Respiratory tract: The inhalation of Marihuana smoke caused bronchial dilation in healthy subjects. Methacholine-induced asthma attacks can be terminated by inhaling marihuana, in this case only psychomimetic cannaboids are active.

Eyes: The ability of cannabis products to reduce intra--ocular pressure was discovered accidentally during trials on the effect of inhaling high doses. During the tests, intra-occular pressure dropped by 45%. Eye drops applied locally had the same effect as standard medication but the effect lasted longer.

Immune system: In vitro and in animal testing, depending on the tissue, the immune system was significantly suppressed after cannaboid administration.

Antimicrobial action: CBC CBDA, CBG and d-9-tetrahydrocannabinol displayed antibacterial effects. CBC and d-9-tetrahydrocannabinol are bacteriostatic and bactericidal against streptocci and staphylococci.

Tumor inhibiting effect: The in-vitro inhibiting effect of d-9-tetrahydrocannabinol, d-8-tetrahydrocannabinol, and CBN on the growth of transplanted lung tumors has been documented.

Heart, circulation: Cannaboids increase heart frequency, peripheral vasodilatation causes an increase in systolic blood pressure in the prone position and a decrease in the supine position.

Other effects: d-9-tetrahydrocannabinol is said to be an appetite stimulant. Long term usage leads to a clear increase in tolerance for most of the pharmacological effects.

Mode of action: Most cannaboids act on the CNS. The multiplicity of effects does not point to just one receptor. Possible interaction with cell-wall lipids or effects on prostoglandin biosynthesis is under discussion at present.

When administered orally, the first psychotropic reactions take effect 30 to 60 minutes later. The effect is at this optimum between 2 to 3 hours later and lasts for a total of 8 hours. When inhaled the effect sets in within a few minutes, reaches its climax or maximum after 30 minutes and lasts for 3 hours.

INDICATIONS AND USAGE

Cannabis was first mentioned in the pharmacopoeia of the Chinese Emperor about 3,000 years ago. Cannabis resin was used for beriberi, constipation, female conditions, gout, malaria, rheumatism and absent-mindedness. In early Indian and Chinese medicine, it was used for nervous depressive states, insomnia, vomiting, tetanus and coughs.

In medieval herbals, it was mostly used externally. There are recipes for balms for healing contractures and for cooling poultices for the head and joints and for podagra.

In 1845, the herb tips were mentioned for internal administration for gonorrhea, angina pectoris and choking fits. It was not until the nineteenth century that Indian hemp was described as having a euphoric effect; it was used for insomnia, neuralgia, painful rheumatism, painful gastrointestinal disorders, cholera, tetanus, epilepsy, strychnine poisoning, acute bronchitis, whooping cough, asthma, impending abortion and weak contractions. The extract was used as a sedative and mild soporific.

Current literature on phytotherapeutic drugs cite as indications for Indian hemp: painful disorders of the alimentary canal such as ulcers or cancer; respiratory disorders such as asthma, emphysema or chronic bronchitis; neuralgia, migraine; urinary tract disorders; mental disorders such as anxiety, neurasthenia or hysteria.

Efficacy has not been proven.

PRECAUTIONS AND ADVERSE REACTIONS

No health hazards or side effects are known in conjunction with the proper administration of designated therapeutic dosages. The intake of toxic dosages, as is common with the smoking of cannabis, leads almost at once to euphoric states (pronounced gaiety, laughing fits) with exaggerated apprehension of sensual impressions. Alterations in the perception of time and space, as well as acoustical, visual and sensory hallucinations, lasting for 2 to 3 hours are common in higher dosages.

Driving ability can be disturbed for as long as 8 hours. Although only rarely reported, acute poisoning symptoms include nausea, vomiting, tear flow, hacking cough, disturbances of cardiac function and numbness of the limbs. Despite its widespread use as a deliriant, instances of death are very rare. The results of chronic abuse are laryngitis, bronchitis, apathy, psychic decline and disturbances of genital functions.

DOSAGE

Mode of Administration: As it is categorized as an illegal narcotic, neither a folk medicinal nor therapeutic usage is officially permitted. It is used illegally as a narcotic. The production of only certain varieties, those with lower levels of 9-tetrahydrocannabinol for the psychotropic effect. More exact dosages are almost impossible to stipulate due to the varieties of action of the d different cannaboids and because of varying breathing techniques.

Daily Dosage: The former average oral single dose of the drug was 0.1 g.
Narcotic: hash and tobacco are mixed. 1 cigarette contains 0.5 g to 1 g of the drug with at least 5 mg to 10 mg d-9-tetrahydrocannabinol for the psychotropic effect. More exact dosages are almost impossible to stipulate due to the varieties of action of the different cannaboids and because of varying breathing techniques.

Storage: Store with care, protected from light. Studies have shown that d-9-tetrahydrocannabinol has a strong affinity with synthetics and rubber and is easily absorbed by them.

LITERATURE.

Anonym, Cannabis: Hanf als Nutzpflanze. In: DAZ 135(27):2538-2541. 1995
Anonym, Rezeptorforschung: Körpereigener Ligand des Cannabis-Rezeptors isoliert. In: DAZ 133(24):2214. 1993.
Clarke CC, Marijuana botany. In: And/Or Press, Berkeley, California. 1981
Drogenmißbrauch: Drogen im Straßenverkehr. In: DAZ 134(27):2575. 1994
Evans, AT el al., (1985) J. Pharm Pharmacol.
Evans AT el al., (1987) FEBS 211: 119.
Evans AT el al., (1987) Biochem Pharmacol 36: 2035.
Evans FJ, Cannabinoids – The separation of central from peripheral effects on a structural basis. In: PM 57:60. 1991.
Fairbairn JW el al., J Pharm Pharmacol 28: 130.
Fairbairn JW, Pickens JT (1981) Br. J. Pharmacol 72: 401.
Gil EW el al., (1970) Nature 228: 135
Goedecke H, Karkos J, Die arzneiliche Verwendung von Cannabisprodukten In: DAZ 136(34):2859-2862 1996.
Jungmayr P, Rauschmittel: Macht Marihuana dumm? In: DAZ 136(34):2867-2868. 1996.
Kovar KA, Cannabis – was ist das? In: DAZ 132(43):2302. 1992.
Nahas, B, In: Marihuana in Science and Medicine. Nahas G (Ed.) Raven Press New York. 1984.
Paris RR el al., (1976) Plant Med Phytother 10:144.
Ross SA, ElSohly MA, The volatile oil composition of fresh and air-dried buds of Cannabis. In: JNP 59(1):49-51. 1996.
Segelman A el al., (1977) J Pharm Sci 66: 1358.
Täschner KL, Drogen und Straßenverkehr. In: DAZ 134(35):3299. 1944.
Turner CE et al., (1980) J Nat Prod 43: 169
Yamaudi T, (1975) Phytochemistry 14: 2189.

Further information in:
Frohne D., Pfänder HJ, Giftpflanzen – Ein Handbuch für Apotheker, Toxikologen und Biologen, 4. Aufl., Wiss, Verlagls-Ges. Stuttgart 1997.
Hänsel R, Keller K, Rimpler H, Schneider G (Hrsg.), Hagers Handbuch der Pharmazeutischen Praxis, 5. Aufl., Bde 4-6 (Drogen), Springer Verlag Berlin, Heidelberg, New York. 1992-1994
Lewin L, Gifte und Vergiftungen, 6. Aufl., Nachdruck, Haug Verlag, Heidelberg 1992.
Madaus G. Lehrbuch der Biologischen Arzneimittel, Bde 1-3, Nachdruck, Georg Olms Verlag Hildesheim 1979.
Roth L, Daunderer M, Kormann K, Giftpflanzen. Pflanzengifte, 4. Aulf., Ecomed Fachverlag Landsberg Lech 1993.
Teuscher E, Lindequist U, Biogene Gifte – Biologie, Chemie, Pharmakologie, 2. Aufl., Fischer Verlag Stuttgart 1994.
Teuscher E, Biogene Arzneimittel, 5. Aufl., Wiss, Verlagsges. Stuttgart 1997.